@article{fdi:010077376,
  title = {{T}rypanosoma brucei gambiense group 2 : the unusual suspect},
  author = {{J}amonneau, {V}incent and {T}ruc, {P}hilippe and {G}r{\'e}baut, {P}ascal and {H}erder, {S}t{\'e}phane and {R}avel, {S}ophie and {S}olano, {P}hilippe and {D}e {M}eeûs, {T}hierry},
  editor = {},
  language = {{ENG}},
  abstract = {{T}rypanosoma brucei causes human {A}frican trypanosomiasis ({HAT}). {T}hree subspecies were described: {T}. b. gambiense ({T}bg) and {T}. b. rhodesiense ({T}br) in humans, and {T}. b. brucei ({T}bb) in animals. {M}olecular markers subdivided {T}bg into two groups: {T}bg1 and {T}bg2, of which the latter is different from {T}bg1 and {T}br (absence of the {SRA} gene), but indistinguishable from {T}bb. {T}bg2 is considered to be a zoonotic form of {HAT} in {W}est {A}frica. {T}bg2 was found mainly in {C}ote d'{I}voire between 1978 and 1992, but the latest description was made in {G}hana in 2013. {N}ew molecular tools would be welcome to characterize such infections and determine their origins (resistance to human serum or patient immunodeficiency) in the current context of {HAT} elimination.},
  keywords = {{AFRIQUE} {SUBSAHARIENNE} ; {AFRIQUE} {DE} {L}'{OUEST} ; {AFRIQUE} {CENTRALE}},
  booktitle = {},
  journal = {{T}rends in {P}arasitology},
  volume = {35},
  numero = {12},
  pages = {983--995},
  ISSN = {1471-4922},
  year = {2019},
  DOI = {10.1016/j.pt.2019.09.002},
  URL = {https://www.documentation.ird.fr/hor/fdi:010077376},
}