@article{fdi:010037857,
  title = {{HIV} type 1 diversity and antiretroviral drug resistance mutations in {B}urundi},
  author = {{V}idal, {N}icole and {N}iyongabo, {T}. and {N}duwimana, {J}. and {B}utel, {C}hristelle and {N}dayiragije, {A}. and {W}akana, {J}. and {N}duwimana, {M}. and {D}elaporte, {E}ric and {P}eeters, {M}artine},
  editor = {},
  language = {{ENG}},
  abstract = {{I}n 2002, an {HIV} surveillance study was performed among more than 5500 individuals representing the general population of urban and rural districts in {B}urundi. {I}n this report, we genetically characterized a subset of the {HIV}-1-positive samples identified during this survey, including all the {HIV}-positive samples from {B}ujumbura, the capital city, and samples from one semiurban and one rural district. {O}ne hundred and nineteen samples were genetically characterized in the {V}3-{V}5 region of the env gene and/or in the protease and reverse transcriptase region of the pol gene. {P}hylogenetic analysis of 101 env/pol sequences revealed that the {HIV}-1 epidemic in {B}urundi was driven by subtype {C} (81.2%), followed by subtype {A} (7.9%) and pol{C}/env{A} recombinants (5.9%). {O}ne major mutation associated with resistance to antiretroviral drugs ({ARV}s) in the pol gene, as defined by the {I}nternational {AIDS} {S}ociety {R}esistance {T}esting-{USA} panel, was observed in one individual, but many minor resistance-associated mutations were also present in the majority of the samples.},
  keywords = {},
  booktitle = {},
  journal = {{A}ids {R}esearch and {H}uman {R}etroviruses},
  volume = {23},
  numero = {1},
  pages = {175--180},
  ISSN = {0889-2229},
  year = {2007},
  DOI = {10.1089/aid.2006.0126},
  URL = {https://www.documentation.ird.fr/hor/fdi:010037857},
}