@article{fdi:010072661,
  title = {{T}he overexpression of {RX}am1, a cassava gene coding for an {RLK}, confers disease resistance to {X}anthomonas axonopodis pv. manihotis},
  author = {{T}atis, {P}. {A}. {D}. and {C}orzo, {M}. {H}. and {C}abezas, {J}. {C}. {O}. and {C}ipagauta, {A}. {M}. and {P}rias, {M}. {A}. and {V}erdier, {V}al{\'e}rie and {A}guirre, {P}. {C}. and {C}arrascal, {C}. {E}. {L}.},
  editor = {},
  language = {{ENG}},
  abstract = {{M}ain conclusion {T}he overexpression of {RX}am1 leads to a reduction in bacterial growth of {X}am{CIO}136, suggesting that {RX}am1 might be implicated in strain-specific resistance. {C}assava bacterial blight ({CBB}) caused by {X}anthomonas axonopodis pv. manihotis ({X}am) is a prevalent disease in all regions, where cassava is cultivated. {CBB} is a foliar and vascular disease usually controlled through host resistance. {P}revious studies have found {QTL}s explaining resistance to several {X}am strains. {I}nterestingly, one {QTL} called {XM}5 that explained 13% of resistance to {X}am{CIO}136 was associated with a similar fragment of the rice {X}a21-resistance gene called {PCR}250. {I}n this study, we aimed to further identify and characterize this fragment and its role in resistance to {CBB}. {S}creening and hybridization of a {BAC} library using the molecular marker {PCR}250 as a probe led to the identification of a receptor-like kinase similar to {X}a21 and were called {RX}am1 ({R} esistance to {X}am 1). {H}ere, we report the functional characterization of susceptible cassava plants overexpressing {RX}am1. {O}ur results indicated that the overexpression of {RX}am1 leads to a reduction in bacterial growth of {X}am{CIO}136. {T}his suggests that {RXAM}1 might be implicated in strain-specific resistance to {X}am{CIO}136.},
  keywords = {{C}assava bacterial blight ; {P}attern-recognition receptor ; {P}lant immunity ; {R} genes ; {R}eceptor-like kinases ; {T}ransgenic cassava},
  booktitle = {},
  journal = {{P}lanta},
  volume = {247},
  numero = {4},
  pages = {1031--1042},
  ISSN = {0032-0935},
  year = {2018},
  DOI = {10.1007/s00425-018-2863-4},
  URL = {https://www.documentation.ird.fr/hor/fdi:010072661},
}