@article{fdi:010059013,
  title = {{H}igh prevalence of hepatitis {E} in humans and pigs and evidence of genotype-3 virus in swine, {M}adagascar},
  author = {{T}emmam, {S}. and {B}esnard, {L}. and {A}ndriamandimby, {S}. {F}. and {F}oray, {C}. and {R}asamoelina-{A}ndriamanivo, {H}. and {H}eraud, {J}. {M}. and {C}ardinale, {E}. and {D}ellagi, {K}oussay and {P}avio, {N}. and {P}ascalis, {H}erv{\'e} and {P}orphyre, {V}.},
  editor = {},
  language = {{ENG}},
  abstract = {{H}epatitis {E} virus ({HEV}) causes an orofecal disease transmitted through poor hygiene environments, contaminated food (mainly pork products), or by contacts with infected animals. {V}ery little data are currently available regarding the disease in the {S}outhwestern {I}ndian {O}cean {I}slands. {W}e report the first sero- and viro-survey for {HEV} in human and swine in {M}adagascar. {A} seroprevalence rate of 14.1% (60 of 427) was measured in slaughterhouse workers. {S}eroprevalence to {HEV} in pigs was estimated to 71.2% (178 of 250), strongly suggesting the existence of a zoonotic cycle. {T}hree out of 250 pig livers (1.2%) tested {HEY} {RNA}-positive by quantitative polymerase chain reaction. {P}hylogenetic analyses based on 1-kb sequences of the {ORF} 2-3 identified these viruses as {HEV} genotype 3. {S}equences clustered in a distinct {M}alagasy sub-clade, possibly representative of a new sub-genotype, for which the date of emergence was estimated around 1989. {F}urther studies are needed to confirm other transmission routes of {HEV} to humans, especially through non-zoonotic cycles.},
  keywords = {{MADAGASCAR}},
  booktitle = {},
  journal = {{A}merican {J}ournal of {T}ropical {M}edicine and {H}ygiene},
  volume = {88},
  numero = {2},
  pages = {329--338},
  ISSN = {0002-9637},
  year = {2013},
  DOI = {10.4269/ajtmh.2012.12-0615},
  URL = {https://www.documentation.ird.fr/hor/fdi:010059013},
}